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One of which is oxidative stress

If translation is initiated from the first start codon, then Glr1 is encoded with a mitochondrial matrix targeting sequence, while translation from the second start codon leads to a shorter cytosolic protein (Outten and Culotta, 2004)

It is thought that substrate reduction is a prerequisite for degradation, and ERAD substrates have long been known to associate with thiol isomerases including ERp90, ERp57, and PDI (Gillece et al., 1999

Hepatic tight-junctional permeability increases following actin-cytoskeleton disruption induced by phalloidin, a toxin that induces actin disorganization by binding specifically to F-actin and impeding its depolymerization.117 As indicated above, F-actin is anchored to tightjunctional-associated proteins (e.g., ZO-1, cingulin), and it is likely that F-actin-conformational disorganization induces relocalization of these intermediary proteins as well